/jp/ - Otaku Culture

[Anonymous No.]

She's beautiful

[Anonymous No.]

That uncanny valley...

[Anonymous No.]

cute

[Anonymous No.]

kike nose

[Anonymous No.]

I actually think milk is cute, but she does not look good with those contacts.

[Anonymous No.]

>>10465054
even worse than the OP image

[Anonymous No.]

http://welcomeunknown.tumblr.com/pt/42557929256

milk a slut

[Anonymous No.]

>>10465052

[Anonymous No.]

Is that the bitch /cgl/ always bitches about?

[Anonymous No.]

What's it made out of?

[Anonymous No.]

[Anonymous No.]

[Anonymous No.]

>by the way, I'm bisexual
https://twitter.com/welcomeUNKNOWNtatus/298887164794650624

milk a slut

[Anonymous No.]

[Anonymous No.]

>>10465062
HEIL

[Anonymous No.]

This is nightmare material

[Anonymous No.]

Is this the same girl?

[Anonymous No.]

Gross.

[STiBoi No.]

>>10465031
I like that pygamy girl better but I respect and appreciate your differing opinion, friend.

[Anonymous No.]

Her face is like the plains of Gorgoroth. A desolate wasteland with vast stretches of land between major features. Her eyes and nose are so far apart she's like the king of Down Syndrome cases.

Plus making shit threads about yourself makes you lose points.

-100000/10

[Anonymous No.]

Quit posting threads about yourself, you dumb whore.

[Anonymous No.]

>>10465086

no

[Anonymous No.]

>>10465107
Shes got dat FAS

[Anonymous No.]

[Anonymous No.]

She looks like a bug

And her eyes are all stretched out its fuckin gross mate

[Anonymous No.]

Don't listen to him milk, I'm pretty sure it's all just one person anyway.

[Anonymous No.]

pls stop bullying ghuise

[Anonymous No.]

>>10465118
>She looks like a bug

Lets get this shit started

[Anonymous No.]

>>10465054
looks like a fucking rat

[Anonymous No.]

you guys got standards too high, that's why you guys will never get laid,

[Anonymous No.]

>>10465067
gross

[Anonymous No.]

>>10465123
>>10465124
>>10465132
You forgot you trip, milky-chan.

[Anonymous No.]

>>10465067
Being "bisexual" is just an excuse to fuck everything that moves.

[Anonymous No.]

>>10465132
Nice try milk-defense-force.

[Anonymous No.]

>>10465067
Fucking disgusting.

[Anonymous No.]

>>10465067
I'd expect better out of even milk

i guess she'll say and do anything in the quest for internet attention

[Anonymous No.]

>>10465141
>>10465145
>>10465140
>>10465136
What more can you expect from a girl who used to be addicted to cocaine and has a half-breed mexican baby?

[Anonymous No.]

>>10465146
Wow, I knew she was a whore but not that bad.

[Anonymous No.]

is that foetal alcohol syndrome? eyes are almost on the side of her head like a cow.

very ugly.

[Anonymous No.]

-Official /jp/ reminder-

If you are currently employed, enrolled in education OR receive a welfare check you do not belong on /jp/. End of story. If you are in the aforementioned categories, please evacuate the premises immediately. Thank you for your cooperation.

[Anonymous No.]

Fetal alcohol exposure is the leading known cause of mental retardation in the Western world.[7][8] In the United States and Europe, the FAS prevalence rate is estimated to be between 0.2-2 in every 1000 live births.[9][10] FAS should not be confused with Fetal Alcohol Spectrum Disorders (FASD), a condition which describes a continuum of permanent birth defects caused by maternal consumption of alcohol during pregnancy, which includes FAS, as well as other disorders, and which affects about 1% of live births in the US.[11][12][13][14] The lifetime medical and social costs of FAS are estimated to be as high as US$800,000 per child born with the disorder.[15] Surveys found that in the United States, 10–15% of pregnant women report having recently drunk alcohol, and up to 30% drink alcohol at some point during pregnancy.[16][17] The current recommendation of the Surgeon General of the United States,the British Department of Health and the Australian Government National Health and Medical Research Council is to drink no alcohol at all during pregnancy.

[Anonymous No.]

Her nose is shaped like a penis.

[Anonymous No.]

>>10465132
In other words, women have their standards for their own behavior very low.

[Anonymous No.]

Fetal alcohol syndrome (FAS) is a pattern of mental and physical defects that can develop in a fetus in association with high levels of alcohol consumption during pregnancy. Alcohol crosses the placental barrier and can stunt fetal growth or weight, create distinctive facial stigmata, damage neurons and brain structures, which can result in psychological or behavioral problems, and cause other physical damage.[1][2][3] [4] The main effect of FAS is permanent central nervous system damage, especially to the brain. Developing brain cells and structures can be malformed or have development interrupted by prenatal alcohol exposure; this can create an array of primary cognitive and functional disabilities (including poor memory, attention deficits, impulsive behavior, and poor cause-effect reasoning) as well as secondary disabilities (for example, predispositions to mental health problems and drug addiction).[3][5] Alcohol exposure presents a risk of fetal brain damage at any point during a pregnancy, since brain development is ongoing throughout pregnancy.

[Anonymous No.]

Growth deficiency is defined as below average height, weight or both due to prenatal alcohol exposure, and can be assessed at any point in the lifespan. Growth measurements must be adjusted for parental height, gestational age (for a premature infant), and other postnatal insults (e.g., poor nutrition), although birth height and weight are the preferred measurements.[11] Deficiencies are documented H191 when height or weight falls at or below the 10th percentile of standardized growth charts appropriate to the patient's population.[22]

The CDC and Canadian guidelines use the 10th percentile as a cut-off to determine growth deficiency.[23][24] The "4-Digit Diagnostic Code" allows for mid-range gradations in growth deficiency (between the 3rd and 10th percentiles) and severe growth deficiency at or below the 3rd percentile.[11] Growth deficiency (at severe, moderate, or mild levels) contributes to diagnoses of FAS and PFAS (Partial Fetal Alcohol Syndrome), but not ARND (Alcohol-Related Neurodevelopmental Disorder) or static encephalopathy.

[Anonymous No.]

>short nose

She isn't a FAS girl, she's just half rat. Isn't she an adult? Isn't it weird to be dressing up in children's clothes trying to look 'cute?'

[Anonymous No.]

Several characteristic craniofacial abnormalities are often visible in individuals with FAS.[25] The presence of FAS facial features indicates brain damage, though brain damage may also exist in their absence. FAS facial features (and most other visible, but non-diagnostic, deformities) are believed to be caused mainly during the 10th and 20th week of gestation.[26]

Refinements in diagnostic criteria since 1975 have yielded three distinctive and diagnostically significant facial features known to result from prenatal alcohol exposure and distinguishes FAS from other disorders with partially overlapping characteristics.[27][28] The three FAS facial features are:
A smooth philtrum — The divot or groove between the nose and upper lip flattens with increased prenatal alcohol exposure.
Thin vermilion — The upper lip thins with increased prenatal alcohol exposure.
Small palpebral fissures — Eye width decreases with increased prenatal alcohol exposure.

Measurement of FAS facial features uses criteria developed by the University of Washington. The lip and philtrum are measured by a trained physician with the Lip-Philtrum Guide,[29] a 5-point Likert Scale with representative photographs of lip and philtrum combinations ranging from normal (ranked 1) to severe (ranked 5). Palpebral fissure length (PFL) is measured in millimeters with either calipers or a clear ruler and then compared to a PFL growth chart, also developed by the University of Washington.

[Anonymous No.]

These distinctive facial features in a patient do strongly correlate to brain damage. Sterling Clarren of the University of Washington's Fetal Alcohol and Drug Unit told a conference in 2002:

“I have never seen anybody with this whole face who doesn't have some brain damage. In fact in studies, as the face is more FAS-like, the brain is more likely to be abnormal. The only face that you would want to counsel people or predict the future about is the full FAS face. But the risk of brain damage increases as the eyes get smaller, as the philtrum gets flatter, and the lip gets thinner. The risk goes up but not the diagnosis.“

“At one-month gestation, the top end of your body is a brain, and at the very front end of that early brain, there is tissue that has been brain tissue. It stops being brain and gets ready to be your face ... Your eyeball is also brain tissue. It's an extension of the second part of the brain. It started as brain and "popped out." So if you are going to look at parts of the brain from alcohol damage, or any kind of damage during pregnancy, eye malformations and midline facial malformations are going to be very actively related to the brain across syndromes ... and they certainly are with FAS.“

[Anonymous No.]

Structural abnormalities of the brain are observable, physical damage to the brain or brain structures caused by prenatal alcohol exposure. Structural impairments may include microcephaly (small head size) of two or more standard deviations below the average, or other abnormalities in brain structure (e.g., agenesis of the corpus callosum, cerebellar hypoplasia).[20]

Microcephaly is determined by comparing head circumference (often called occipitofrontal circumference, or OFC) to appropriate OFC growth charts.[22] Other structural impairments must be observed through medical imaging techniques by a trained physician. Because imaging procedures are expensive and relatively inaccessible to most patients, diagnosis of FAS is not frequently made via structural impairments, except for microcephaly.

Evidence of a CNS structural impairment due to prenatal alcohol exposure will result in a diagnosis of FAS, and neurological and functional impairments are highly likely.

[Anonymous No.]

Central nervous system (CNS) damage is the primary feature of any Fetal Alcohol Spectrum Disorder (FASD) diagnosis. Prenatal exposure to alcohol — which is classified as a teratogen — can damage the brain across a continuum of gross to subtle impairments, depending on the amount, timing, and frequency of the exposure as well as genetic predispositions of the fetus and mother.[20][33] While functional abnormalities are the behavioral and cognitive expressions of the FAS disability, CNS damage can be assessed in three areas: structural, neurological, and functional impairments.

All four diagnostic systems allow for assessment of CNS damage in these areas, but criteria vary. The IOM system requires structural or neurological impairment for a diagnosis of FAS.[20] The "4-Digit Diagnostic Code" and CDC guidelines state that functional anomalies must measure at two standard deviations or worse in three or more functional domains for a diagnosis of FAS.[11][23] The "4-Digit Diagnostic Code" further elaborates the degree of CNS damage according to four ranks:
Definite — Structural impairments or neurological impairments for FAS or static encephalopathy.
Probable — Significant dysfunction of two standard deviations or worse in three or more functional domains.
Possible — Mild to moderate dysfunction of two standard deviations or worse in one or two functional domains or by judgment of the clinical evaluation team that CNS damage cannot be dismissed.
Unlikely — No evidence of CNS damage.

[Anonymous No.]

When structural or neurological impairments are not observed, all four diagnostic systems allow CNS damage due to prenatal alcohol exposure to be assessed in terms of functional impairments.[20][11][23][24] Functional impairments are deficits, problems, delays, or abnormalities due to prenatal alcohol exposure (rather than hereditary causes or postnatal insults) in observable and measurable domains related to daily functioning, often referred to as developmental disabilities. There is no consensus on a specific pattern of functional impairments due to prenatal alcohol exposure[20] and only CDC guidelines label developmental delays as such,[23] so criteria vary somewhat across diagnostic systems.

The four diagnostic systems list various CNS domains that can qualify for functional impairment that can determine an FAS diagnosis:
Evidence of a complex pattern of behavior or cognitive abnormalities inconsistent with developmental level in the following CNS domains — sufficient for a PFAS (partial fetal alcohol syndrome) or ARND (alcohol-related neurodevelopmental disorder) diagnosis using IOM guidelines[20]
Learning disabilities, academic achievement, impulse control, social perception, communication, abstraction, math skills, memory, attention, judgment
Performance at two or more standard deviations on standardized testing in three or more of the following CNS domains — sufficient for a FAS, PFAS or static encephalopathy diagnosis using 4-Digit Diagnostic Code[11]
Executive functioning, memory, cognition, social/adaptive skills, academic achievement, language, motor skills, attention, activity level

[Anonymous No.]

>>10465151
>britfag calling someone ugly
lol

[Anonymous No.]

>>10465168
The short FAS nose is cancelled by her jewish heritage

[Anonymous No.]

During the first trimester of pregnancy, alcohol interferes with the migration and organization of brain cells, which can create structural deformities or deficits within the brain.[34] During the third trimester, damage can be caused to the hippocampus, which plays a role in memory, learning, emotion, and encoding visual and auditory information, all of which can create neurological and functional CNS impairments as well.[35]

As of 2002, there were 25 reports of autopsies on infants known to have FAS. The first was in 1973, on an infant who died shortly after birth.[36] The examination revealed extensive brain damage, including microcephaly, migration anomalies, callosal dysgenesis, and a massive neuroglial, leptomeningeal heterotopia covering the left hemisphere.[37]

In 1977, Dr. Clarren described a second infant whose mother was a binge drinker. The infant died ten days after birth. The autopsy showed severe hydrocephalus, abnormal neuronal migration, and a small corpus callosum (which connects the two brain hemispheres) and cerebellum.[37] FAS has also been linked to brainstem and cerebellar changes, agenesis of the corpus callosum and anterior commissure, neuronal migration errors, absent olfactory bulbs, meningomyelocele, and porencephaly.

[Anonymous No.]

is this cuter milk sister

[Anonymous No.]

-Official /jp/ reminder-

If you are currently employed, enrolled in education OR receive a welfare check you do not belong on /jp/. End of story. If you are in the aforementioned categories, please evacuate the premises immediately. Thank you for your cooperation.

[Anonymous No.]

>>10465177
disgusting eyebrows

[Anonymous No.]

General cognitive deficits (e.g., IQ) at or below the 3rd percentile on standardized testing — sufficient for an FAS diagnosis using CDC guidelines[23]
Performance at or below the 16th percentile on standardized testing in three or more of the following CNS domains — sufficient for an FAS diagnosis using CDC guidelines[23]
Cognition, executive functioning, motor functioning, attention and hyperactive problems, social skills, sensory integration dysfunction, social communication, memory, difficulties responding to common parenting practices
Performance at two or more standard deviations on standardized testing in three or more of the following CNS domains — sufficient for an FAS diagnosis using Canadian guidelines
Cognition, communication, academic achievement, memory, executive functioning, adaptive behavior, social skills, social communication

Other conditions may commonly co-occur with FAS, stemming from prenatal alcohol exposure. However, these conditions are considered Alcohol-Related Birth Defects[20] and not diagnostic criteria for FAS.
Cardiac — A heart murmur that frequently disappears by one year of age. Ventricular septal defect most commonly seen, followed by an atrial septal defect.
Skeletal — Joint anomalies including abnormal position and function, altered palmar crease patterns, small distal phalanges, and small fifth fingernails.
Renal — Horseshoe, aplastic, dysplastic, or hypoplastic kidneys.
Ocular — Strabismus, optic nerve hypoplasia[38] (which may cause light sensitivity, decreased visual acuity, or involuntary eye movements).
Occasional abnormalities — ptosis of the eyelid, microophthalmia, cleft lip with or without a cleft palate, webbed neck, short neck, tetralogy of Fallot, coarctation of the aorta, spina bifida, and hydrocephalus.

[Anonymous No.]

Prenatal alcohol exposure is the cause of fetal alcohol syndrome. A study of over 400,000 American women, all of whom had consumed alcohol during pregnancy, concluded that consumption of 15 drinks or more per week was associated with a reduction in birth weight.[39] Though consumption of less than 15 drinks per week was not proven to cause FAS-related effects, the study authors recommend limiting consumption to no more than one standard drink per day.[39] Also, threshold values are based upon group averages, and it is not appropriate to conclude that exposure below this threshold is necessarily ‘safe’ because of the significant individual variations in alcohol pharmacokinetics.[39]

An analysis of seven medical research studies involving over 130,000 pregnancies found that consuming two to 14 drinks per week did not significantly increase the risk of giving birth to a child with either malformations or fetal alcohol syndrome.[40] Pregnant women who consume approximately 18 drinks per day have a 30-33% chance of having a baby with FAS.

[Anonymous No.]

Ugly cowcasian bitch.

[Anonymous No.]

>>10465177
Eyebrows are going to join with her hairline before long

[Anonymous No.]

Amount, frequency, and timing of prenatal alcohol use can dramatically impact the other three key features of FAS. While consensus exists that alcohol is a teratogen, there is no clear consensus as to what level of exposure is toxic.[20] The CDC guidelines are silent on these elements diagnostically. The IOM and Canadian guidelines explore this further, acknowledging the importance of significant alcohol exposure from regular or heavy episodic alcohol consumption in determining, but offer no standard for diagnosis. Canadian guidelines discuss this lack of clarity and parenthetically point out that "heavy alcohol use" is defined by the National Institute on Alcohol Abuse and Alcoholism as five or more drinks per episode on five or more days during a 30 day period.[47]

"The 4-Digit Diagnostic Code" ranking system distinguishes between levels of prenatal alcohol exposure as High Risk and Some Risk. It operationalizes high risk exposure as a blood alcohol concentration (BAC) greater than 100 mg/dL delivered at least weekly in early pregnancy. This BAC level is typically reached by a 55 kg woman drinking six to eight beers in one sitting.

[Anonymous No.]

Fetal alcohol syndrome is the only expression of FASD that has garnered consensus among experts to become an official ICD-9 and ICD-10 diagnosis. To make this diagnosis (or determine any FASD condition), a multi-disciplinary evaluation is necessary to assess each of the four key features for assessment. Generally, a trained physician will determine growth deficiency and FAS facial features. While a qualified physician may also assess central nervous system structural abnormalities and/or neurological problems, usually central nervous system damage is determined through psychological assessment. A pediatric neuropsychologist may assess all areas of functioning, including intellectual, language processing, and sensorimotor. Prenatal alcohol exposure risk may be assessed by a qualified physician or psychologist.

The following criteria must be fully met for an FAS diagnosis:[20][11][23][24]
Growth deficiency — Prenatal or postnatal height or weight (or both) at or below the 10th percentile[22]
FAS facial features — All three FAS facial features present[30]
Central nervous system damage — Clinically significant structural, neurological, or functional impairment
Prenatal alcohol exposure — Confirmed or Unknown prenatal alcohol exposure

[Anonymous No.]

Traditional behavioral interventions are predicated on learning theory, which is the basis for many parenting and professional strategies and interventions.[49] Along with ordinary parenting styles, such strategies are frequently used by default for treating those with FAS, as the diagnoses Oppositional Defiance Disorder (ODD), Conduct Disorder, Reactive Attachment Disorder (RAD), etc. often overlap with FAS (along with ADHD), and these are sometimes thought to benefit from behavioral interventions. Frequently, a patient's poor academic achievement results in special education services, which also utilizes principles of learning theory, behavior modification, and outcome-based education.

Because the "learning system" of a patient with FAS is damaged, however, behavioral interventions are not always successful, or not successful in the long run, especially because overlapping disorders frequently stem from or are exacerbated by FAS.[49] Kohn (1999) suggests that a rewards-punishment system in general may work somewhat in the short term but is unsuccessful in the long term because that approach fails to consider content (i.e., things "worth" learning), community (i.e., safe, cooperative learning environments), and choice (i.e., making choices versus following directions).[50] While these elements are important to consider when working with FAS and have some usefulness in treatment, they are not alone sufficient to promote better outcomes.[49] Kohn's minority challenge to behavioral interventions does illustrate the importance of factors beyond learning theory when trying to promote improved outcomes for FAS, and supports a more multi-model approach that can be found in varying degrees within the advocacy model and neurobehavioral approach.

[Anonymous No.]

The primary disabilities of FAS are the functional difficulties with which the child is born as a result of CNS damage due to prenatal alcohol exposure.[5] Often, primary disabilities are mistaken as behavior problems, but the underlying CNS damage is the originating source of a functional difficulty,[54] rather than a mental health condition, which is considered a secondary disability.

The exact mechanisms for functional problems of primary disabilities are not always fully understood, but animal studies have begun to shed light on some correlates between functional problems and brain structures damaged by prenatal alcohol exposure.[3] Representative examples include:
Learning impairments are associated with impaired dendrites of the hippocampus[55]
Impaired motor development and functioning are associated with reduced size of the cerebellum[56]
Hyperactivity is associated with decreased size of the corpus callosum

[Anonymous No.]

Functional difficulties may result from CNS damage in more than one domain, but common functional difficulties by domain include:[3][49][51][54] Note that this is not an exhaustive list of difficulties.
Achievement — Learning disabilities
Adaptive behavior — Poor impulse control, poor personal boundaries, poor anger management, stubbornness, intrusive behavior, too friendly with strangers, poor daily living skills, developmental delays
Attention — Attention-Deficit/Hyperactivity Disorder (ADHD), poor attention or concentration, distractible
Cognition — Mental retardation, confusion under pressure, poor abstract skills, difficulty distinguishing between fantasy and reality, slower cognitive processing
Executive functioning — Poor judgment, Information-processing disorder, poor at perceiving patterns, poor cause and effect reasoning, inconsistent at linking words to actions, poor generalization ability
Language — Expressive or receptive language disorders, grasp parts but not whole concepts, lack understanding of metaphor, idioms, or sarcasm
Memory — Poor short-term memory, inconsistent memory and knowledge base
Motor skills — Poor handwriting, poor fine motor skills, poor gross motor skills, delayed motor skill development (e.g., riding a bicycle at appropriate age)
Sensory integration and soft neurological problems — sensory integration dysfunction, sensory defensiveness, undersensitivity to stimulation
Social communication — Intrude into conversations, inability to read nonverbal or social cues, "chatty" but without substance

[Anonymous No.]

>>10465184
Eyebrows like that are cute.

[Anonymous No.]

The secondary disabilities of FAS are those that arise later in life secondary to CNS damage. These disabilities often emerge over time due to a mismatch between the primary disabilities and environmental expectations; secondary disabilities can be ameliorated with early interventions and appropriate supportive services.[5]

Six main secondary disabilities were identified in a University of Washington research study of 473 subjects diagnosed with FAS, PFAS (partial fetal alcohol syndrome), and ARND (alcohol-related neurodevelopmental disorder):[3][5]
Mental health problems — Diagnosed with ADHD, Clinical Depression, or other mental illness, experienced by over 90% of the subjects
Disrupted school experience — Suspended or expelled from school or dropped out of school, experienced by 60% of the subjects (age 12 and older)
Trouble with the law — Charged or convicted with a crime, experienced by 60% of the subjects (age 12 and older)
Confinement — For inpatient psychiatric care, inpatient chemical dependency care, or incarcerated for a crime, experienced by about 50% of the subjects (age 12 and older)
Inappropriate sexual behavior — Sexual advances, sexual touching, or promiscuity, experienced by about 50% of the subjects (age 12 and older)
Alcohol and drug problems — Abuse or dependency, experienced by 35% of the subjects (age 12 and older)

[Anonymous No.]

We get it, spergo. She has FAS.

[Anonymous No.]

Two additional secondary disabilities exist for adult patients:

Dependent living — Group home, living with family or friends, or some sort of assisted living, experienced by 80% of the subjects (age 21 and older)

Problems with employment — Required ongoing job training or coaching, could not keep a job, unemployed, experienced by 80% of the subjects (age 21 and older)

[Anonymous No.]

Eight factors were identified in the same study as universal protective factors that reduced the incidence rate of the secondary disabilities:[3][5]
Living in a stable and nurturing home for over 73% of life
Being diagnosed with FAS before age six
Never having experienced violence
Remaining in each living situation for at least 2.8 years
Experiencing a "good quality home" (meeting 10 or more defined qualities) from age 8 to 12 years old
Having been found eligible for developmental disability (DD) services
Having basic needs met for at least 13% of life
Having a diagnosis of FAS (rather than another FASD condition)

[Anonymous No.]

milk a slut